U.S. Scientists Find Cell that Attacks HIV Virus

var gaJsHost = (("https:" == document.location.protocol) ? "https://ssl." : "http://www."); document.write(unescape("%3Cscript src='" + gaJsHost + "google-analytics.com/ga.js' type='text/javascript'%3E%3C/script%3E")); ion" CONTENT="U.S. scientists announced on Thursday that the discovery of immune cells that attack HIV and could be a potential cure for AIDS. Dr. Jay Levy and collaborators found that the immune cells, called CDT-T, decrease levels of the virus in the blood, causing the flu-like illness experienced during the first stages of AIDS t"> U.S. Scientists Find Cell that Attacks HIV Virus < > // var gaJsHost = (("https:" == document.location.protocol) ? "https://ssl." : "http://www."); document.write(unescape("%3Cscript src='" + gaJsHost + "google-analytics.com/ga.js' type='text/javascript'%3E%3C/script%3E")); > < > // var gaJsHost = (("https:" == document.location.protocol) ? "https://ssl." : "http://www."); document.write(unescape("%3Cscript src='" + gaJsHost + "google-analytics.com/ga.js' type='text/javascript'%3E%3C/script%3E")); >

Important note: Information in this article was accurate in 1994. The state of the art may have changed since the publication date.

U.S. Scientists Find Cell that Attacks HIV Virus

Reuters (12/15/94)

U.S. scientists announced on Thursday that the discovery of immune cells that attack HIV and could be a potential cure for AIDS. Dr. Jay Levy and collaborators found that the immune cells, called CDT-T, decrease levels of the virus in the blood, causing the flu-like illness experienced during the first stages of AIDS to subside after two to six weeks. The CDT-T cells appeared to briefly stop the spread of the disease. The University of California at San Francisco researchers concluded that the discovery of the HIV-attacking cells will help in understanding the disease. The study found that three out of seven patients had reduced HIV levels in their blood as their blood immune cell activity increased. The researchers said that the finding suggests the cells were "responsible for the control of HIV replication."

941216
AD942248

Copyright © 1994 - Information, Inc., Bethesda, MD. The CDC National Center for HIV, STD and TB Prevention provides the following information as a public service only. Providing synopses of key scientific articles and lay media reports on HIV/AIDS, other sexually transmitted diseases and tuberculosis does not constitute CDC endorsement. This daily update also includes information from CDC and other government agencies, such as background on Morbidity and Mortality Weekly Report (MMWR) articles, fact sheets, press releases and announcements. Reproduction of this text is encouraged; however, copies may not be sold, and the CDC HIV/STD/TB Prevention News Update should be cited as the source of the information. Contact the sources of the articles abstracted below for full texts of the articles.

AEGiS is made possible through unrestricted funding from Boehringer Ingelheim, Elton John AIDS Foundation, iMetrikus, Inc., John M. Lloyd Foundation, the National Library of Medicine, and donations from users like you. Always watch for outdated information. This article first appeared in 1994. This material is designed to support, not replace, the relationship that exists between you and your doctor.

AEGiS presents published material, reprinted with permission and neither endorses nor opposes any material. All information contained on this website, including information relating to health conditions, products, and treatments, is for informational purposes only. It is often presented in summary or aggregate form. It is not meant to be a substitute for the advice provided by your own physician or other medical professionals. Always discuss treatment options with a doctor who specializes in treating HIV.

Copyright ©1980, 1994. AEGiS. All materials appearing on AEGiS are protected by copyright as a collective work or compilation under U.S. copyright and other laws and are the property of AEGiS, or the party credited as the provider of the content. .